The pathobiology of neurovascular aging

As the population ages, neurovascular pathologies have become leading causes of death and disability. Santisteban and Iadecola review data showing that neurovascular aging not only reduces cerebral blood flow but also impairs protein clearance, immune function, and neurotrophic homeostasis, thereby increasing the risk of stroke, vascular dementia, and Alzheimer’s disease.

How our cells dispose of waste and ways to control it

Cells degrade components that are no longer needed through autophagy. New results show that a weak molecular interaction is essential for this process. By modifying this interaction, it is possible to artificially trigger autophagy, which could then enable the degradation of deposits in neurodegenerative diseases such as Alzheimer’s, or support cancer therapies.

A novel multipotent conjugate bearing tacrine and donepezil motifs with dual cholinergic inhibition and neuroprotective properties targeting Alzheimer’s disease

In this work, we had developed potential multifunctional agents to combat Alzheimer´s disease. Our strategy merge fragments of tacrine and donepezil in a unique hybrid structure. These compounds were synthetized successfully, and then evaluated for their dual AChE/BuChE inhibitor potential and neuroprotector response using a glutamate-induced excitotoxicity model. Most of compounds showed promising activity. Among… Continue reading A novel multipotent conjugate bearing tacrine and donepezil motifs with dual cholinergic inhibition and neuroprotective properties targeting Alzheimer’s disease

Breakthrough in protein homeostasis offers potential for preventing Alzheimer’s

Scientists have uncovered a powerful ally in the fight against neurodegenerative diseases: a nucleolar complex that plays a pivotal role in maintaining cellular health through protein homeostasis (proteostasis), by which cells maintain the balance and proper functioning of their proteins. By suppressing this complex, researchers have shown it’s possible to dramatically reduce the toxic effects… Continue reading Breakthrough in protein homeostasis offers potential for preventing Alzheimer’s

Gantenerumab in Dominantly Inherited Alzheimer Disease—Reply

In Reply Pomara and Imbimbo raise important points in their letter regarding our recent publication. Specifically, they offer additional considerations for the increases of the soluble form of triggering receptor expressed on myeloid cells 2 (sTREM2) in participants receiving gantenerumab compared with placebo. We appreciate the chance to address the points raised.