DOT1L-mediated RAP80 methylation promotes BRCA1 recruitment to elicit DNA repair

AI Summary

This article discusses how DOT1L-mediated methylation of RAP80 promotes the recruitment of BRCA1 for DNA repair processes. This mechanism plays a crucial role in maintaining genomic stability and preventing the development of cancer. The findings offer insights into potential therapeutic targets for enhancing DNA repair mechanisms in cancer cells.

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